H. pylori
Point-of-care rapid testing for h. pylori. OEM/ODM manufacturer.
Overview
Helicobacter pylori colonizes the gastric mucosa and is linked to gastritis, peptic ulcer and gastric cancer. Non-invasive testing supports test-and-treat strategies.
Diagnostic Markers & Methods
Stool antigen (active infection) · serology (antibody, exposure) · urea breath test (reference non-invasive).
Testing Window
Stool antigen detectable during active colonization; PPI/antibiotic washout before testing.
Interpretation
Stool antigen detects active infection and is suitable for post-treatment confirmation; serology cannot distinguish active from past infection.
Confirmatory Testing
Urea breath test or endoscopy with biopsy.
The Author's Take
Position: Prefer the stool antigen test over serology for H. pylori — it detects active infection and works for test-of-cure, which serology cannot.
- Antibody tests stay positive after treatment; stool antigen confirms eradication.
- Test-and-treat decisions need active-infection detection, not exposure history.
- PPI/antibiotic timing affects stool antigen — put that caveat on your datasheet.
This is the buyer-advisor view of the technical team — editorial guidance, not a substitute for product IFU or local regulatory requirements.
Frequently Asked Questions
Which H. pylori test is best for test-of-cure?
Stool antigen (or urea breath test) — they detect active infection and can confirm eradication. Antibody tests stay positive after treatment and are unsuitable for test-of-cure.
Do antibiotics or PPIs affect H. pylori stool antigen?
Yes — recent PPI or antibiotic use can suppress detection and cause false negatives; testing is usually done after a washout period.
Is H. pylori serology useful?
It indicates exposure but cannot distinguish active from past infection; use it mainly where stool testing or endoscopy is unavailable.
Products (2)
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